A Natural Environmental Health Facts Ezine

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"A NATURAL ENVIRONMENTAL HEALTH FACTS Ezine" Here to Inform and Help You Become Healthier and Happier while Achieving Quality Longevity!

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Wednesday July 20, 2005
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=> IN THIS ISSUE!
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==> Editors' Ranting & or Warnings
==> Something To Think About
==> Health Thought for the day!
==> Showcase Health Spotlight
==> Monthly Spotlight Ads
==> Today's Health Tip
==> Food of The Week
==> Health Today
==> Environmental Report
==> Life Changing Information

Today, as usual, I'm giving you some thought provoking and in some cases controversial information in regards to your health... Enjoy...

As lightening caused fires continue and the smoke envelopes us I will be in the valley for the day, escaping some of it... I will have my cell phone with me and if you need to reach me call, if it can wait leave a message and I will get back to you tomorrow...

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Take charge of you and your family's health before it takes charge of you!
Lena

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Something To Think About
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In May, 2005, scientists at Louisiana State University showed that black raspberries contain antiangiogenic compounds that are capable of restraining tumor growth. Antiangiogenic compounds work by inhibiting the formation of new blood vessels, without which tumors cannot expand. The Baton Rouge researchers discovered that berries contain a "highly potent antiangiogenic fraction that accounts for only one percent of the fresh weight of whole black raspberries." The scientists consider it natural and potent enough to use clinically as a "promising complementary cancer therapy" (Liu 2005).

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We accept all advertisements in good faith, but the advertisers are completely responsible for the content and accuracy of their advertisements. We do not give any warranties and accept no responsibility. The editor and publisher suggest that you exercise due diligence!
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Feeding Ulcers!

According to a recent Polish study presented at this past spring's 2005 Digestive Disease Week conference in Chicago, grapefruit seed extract may also help to heal stomach ulcers. Lab rats experienced a 50% reduction in ulcer-causing gastric acid secretion, and progressive decreases in ulcer size following either 6 or 9 days of treatment. The study also noted a major increase in blood flow at ulcer sites.

Interestingly enough, it was the antioxidant properties of the extract that were touted by the study's authors as the active component. No mention was made of the well-known antibacterial effects of grapefruit seed extract, one of the most powerful natural bacteria-killers in the world. I wonder if they considered this, or if the study simply proved that the antioxidants were responsible.

Either way, if you suffer from stomach ulcers, you may want to give some grapefruit seed extract a try. You can get it most anywhere - health food stores and compounding pharmacies are some of the best sources.

My mother would tell me the story; when I was 18 months old I ate a half can of lye, that she was using to make soap. This ate the lining of my esophagus and left my upper passages looking like a web that spiders had built, according to the doctor who read my esophagram a few years ago. But shortly after that incident while my throat was still very raw my mother said she would find me sitting in the garden eating ripe tomatoes and crying from the pain, but she was unable to convince me that I should not eat them. Guess that was when my love affair with tomatoes began...

Tomatoes continue to be one of my favorite foods into this ripe old-age of 68! I now know that they are very good for me and you!

Tomatoes generally range in size from three-quarters of an inch in diameter and a quarter of an ounce in weight to six inches in diameter and two pounds. Tomatoes range in hue from white to red to purple, green yellow and orange. Generally, the lighter colored tomatoes have a milder taste. The red colored tomatoes get their red pigment due to the lycopene content. More on lycopene later on…

If you're like most people, you think tomatoes are vegetable. If you talk to a botanist, he or she might insist that the tomato is a fruit because it contains seeds with sweet pulp. However, a horticulturist would argue that the tomato plant is a vegetable plant and not like fruits that are grown on trees (eg. apple, peach, pear, etc.). Both arguments are valid, and in a way, the answer is most likely tomatoes are both a fruit and a vegetable.

Tomatoes are the second most consumed vegetable in North America. The tomato is a treasure of nutrients, including Vitamin C, Vitamin A, potassium, folic acid and more. Processed tomatoes have higher levels of the nutrients simply because it is concentrated. North Americans customarily eat a large volume of tomatoes and tomato products that it ranks as No.1 among fruits and vegetables in the total amount of nutrients they contribute to the diet.

Interestingly, second to oranges, tomatoes contribute a high amount of Vitamin C in North American diets. Vitamin C has long been known to prevent scurvy - a condition that is characterized by bleeding gums and soreness in the joints. Vitamin C is also known to function as an antioxidant in the human body. Studies have shown that it may prevent certain degenerative diseases and common colds.

In addition, tomato consumption is listed as the fourth most important source of vitamin A. In the body, vitamin A is converted from sources beta-carotene and it is important for immune functions, maintenance of the skin and tissue lining and vision.

Potassium is an essential nutrient that is important for normal health maintenance and growth. It is required for movement of nutrients across the cells in the body. A deficiency of potassium will lead to muscle weakness, causing heart palpitations, heart attack or heart disease, apathy, cramps and more. It is important for athletes to maintain levels of potassium which is lost through sweating during an event. Potassium levels can be easily maintained before or after the event by eating foods or drinking fluids high in potassium.

Tomatoes were first thought to be poison and were not to be eaten but that idea lost it's wind and then became the food of necessity.

The tomato originated in the Andes Mountains in South America and was first domesticated in Mexico by the Aztec people. After the Spanish conquered Mexico in the early 1500s, they introduced it to southern Europe, where it quickly became popular. The Italians named it "poma amoris" meaning "apple of love". Similarly, the French also called it "pomme d'amour". This name gave rise to the legend that the tomato was an aphrodisiac. However, in colder climates, such as Canada, Great Britain and parts of the United States, it was called the "wolf peach". This unappealing name reflected its poor reputation and people's beliefs that tomatoes were poisonous. Therefore, for many years, the tomato plant was only grown for decorative purposes.

The most talked about food nutrient known, at this time in the tomato, is Lycopene and being studied for further health benefits. In addition to giving tomatoes their vibrant color, lycopene appears to protect against diseases such as cancer and heart disease.

Lycopene is an “antioxidant” - it helps to counteract the harmful effects of substances called “free radicals,” which are thought to contribute to many chronic diseases and age-related processes in the body.

The researchers surveyed the eating habits of over 47,000 men between the ages of 40-75 for six years and found that the consumption of tomatoes, tomato sauce, tomato juice and pizza was associated with a reduced risk for developing prostate cancer. Researchers theorize that lycopene, an antioxidant nutrient found in large amounts in tomatoes, may be responsible for this possible protective effect.

I serve up hug plates of sliced fresh tomatoes with sliced cucumbers splashed with a bit of rice vinegar, salt and pepper and the family loves it. My dh grows our own organic tomatoes with which I make our ketchup, tomato sauce and canned fresh tomatoes to go with us through the winter. Try a small patch, as tomatoes are easily grown in your flower bed!

Tomato season is coming into full swing here, if that is the case in your area go out and get some great organically grown tomatoes for maximum health and nutrient content.

'Double Diabetes' Harder to Detect, Treat
Jul 19, 2005
By LAURAN NEERGAARD

WASHINGTON (AP) - Having one type of diabetes is bad enough, but two? Doctors are seeing a new phenomenon dubbed double diabetes that makes it harder to diagnose and treat patients - especially children.

The mix can strike at any age, and comes in various forms: Children who depend on insulin injections because of Type 1 diabetes gain weight and then get the Type 2 form in which their bodies become insulin resistant, for example.

Or someone with classic Type 2 symptoms isn't responding to therapy, and tests reveal they also are developing the insulin-dependent form of the disease. Or they may not fall clearly into either category.

The labels are important - different forms require different treatments.

Yet "there are many people in which it's very blurred as to what kind of diabetes they have," says Dr. Francine Kaufman, a University of Southern California pediatric endocrinologist and past president of the American Diabetes Association.

There are no good statistics on this complex disease-mixing.

But the Children's Hospital of Pittsburgh counts about 25 percent of child patients with Type 1 diabetes who also are overweight and have other Type 2 features, says Dr. Dorothy Becker, a pediatric endocrinologist and leading double-diabetes researcher.

And an ongoing study to determine the best treatment for child Type 2 diabetics is uncovering many participants who harbor antibodies that signal they have or are developing the Type 1 form, too, says Kaufman.

Those findings echo a handful of recent research reports raising concern about the phenomenon, which some call atypical diabetes or "diabetes 1 1/2" or even Type 3 diabetes.

Diabetes occurs when the body can't turn blood sugar, or glucose, into energy, either because it doesn't produce enough insulin or doesn't use it correctly.

With the Type 1 form, the patient's own immune system attacks the insulin-producing islet cells in the pancreas. Once thought to strike only in childhood, it also can develop in adults. Symptoms usually appear suddenly and can quickly become life-threatening. Insulin, given by shots or a pump, is required to survive.

With the Type 2 form, the body loses its ability to use insulin properly, even though the pancreas pumps out extra and drugs often are given to rev up that production even more. Type 2 usually develops slowly, and once was thought to hit only the middle-aged but now is striking even overweight children.

Both forms can lead to heart and kidney disease, blindness and amputations, and kill if not properly treated. But Type 2, which afflicts over 90 percent of the more than 18 million U.S. diabetics, has gotten more attention recently because it's an epidemic fueled by increasing obesity.

Yet specialists knew Type 1 was quietly increasing, too - and then they began spotting double diabetics.

The theory: Overweight people need more insulin to process glucose regardless of whether they're insulin-resistant yet. So, perhaps obesity overworks the pancreas until it wears out, Pittsburgh's Becker suggests. Or perhaps obesity accelerates the autoimmune destruction - meaning someone genetically predisposed to Type 1 diabetes might not have gotten it had they stayed thin.

"You've not just exceeded what you can make but perhaps accelerated the destruction," and then insulin-resistance sets in, agrees Kaufman, who just authored a book called "Diabesity" exploring the overall obesity-diabetes threat.

Whatever you call that mix, it complicates treatment.

Consider Martha Larkin of Pittsburgh, diagnosed with Type 1 diabetes at age 3. For years, her mother would wake up in the middle of the night to test Martha's blood sugar and administer insulin. Set mealtimes and off-limit foods became the family's norm.

Then early puberty hit at 10, and Martha began gaining weight, says her mother, Cindy Stevans. Now almost 12, Martha's daily insulin requirement grew to that of grown man, signaling developing insulin resistance. And, in a vicious cycle, the more insulin she gets, the hungrier she feels.

A recently implanted insulin pump is helping, and the family joined a pool in hopes that physical activity will help Martha stave off double diabetes - and that her twin brother will stay diabetes-free. But weight is a problem for this whole family of bookworms who hate exercise so much that Stevans calls it "torture."

"It's painfully hard," she says of her daughter's co-battles with diabetes and weight.

Scientists don't yet know if double diabetics will need special treatments. For now, the emphasis is on prevention. For Type 2, that means weight loss. For Type 1, scientists are enrolling pregnant women from diabetes-prone families into a major study to hunt what might protect their babies from the illness later in life. To enroll, check .http://www.trigr.org

LENA'S NOTE - I'm not saying put yourself up for being a guinea pig but the information is here to use as you see fit...

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GENETICALLY MODIFIED CORN STUDY REVEALS HEALTH DAMAGE & COVER-UP
By Jeffrey Smith
July 16, 2005

When a German court ordered Monsanto to make public a controversial 90-day rat study on June 20, 2005, the data upheld claims by prominent scientists who said that animals fed the genetically modified (GM) corn developed extensive health effects in the blood, kidneys and liver and that humans eating the corn might be at risk. The 1,139 page research paper on Monsanto?s ?Mon 863? variety also revealed that European regulators accepted the company?s assurances that their corn is safe, in spite of the unscientific and contradictory rationale that was used to dismiss significant problems. In addition, the study is so full of flaws and omissions, critics say it wouldn?t qualify for publication in most journals and yet it is the primary document used to evaluate the health impacts.

Mon 863 is genetically engineered to produce a form of a pesticide called bacillus thuringiensis or Bt, designed to attack a corn pest called the root worm. Rats fed Mon 863 developed several reactions, including those typically found with allergies (increased basophils), in response to infections, toxins and various diseases including cancer (increased lymphocytes and white blood cells), and in the presence of anemia (decreased reticulocyte count) and blood pressure problems (decreased kidney weights). There were also increased blood sugar levels, kidney inflammation, liver and kidney lesions, and other changes. According to top research biologist Arpad Pusztai, who was commissioned by the German government to evaluate the study in 2004, based on the evidence no one can say that Mon 863 will cause cancer or allergies or anything specific. The results are preliminary and must be followed-up to rule these out. He warns, however, ?It is almost impossible to imagine that major lesions in important organs. . . . or changes in blood parameters. . . . that occurred in GM maize-fed rats, is incidental and due to simple biological variability."

French Professor Gilles-Eric Seralini, a molecular endocrinologist at the University of Caen, agrees that the results indicate a toxic reaction. Seralini is a member of two French government commissions that evaluate GM food, one of which originally rejected a request for approval of the corn variety in October, 2003 due to the adverse findings of the study. Seralini won a French lawsuit allowing him to express his concerns in public, and now Greenpeace has won a German court battle that makes public the data that is the source of his concerns.

Pusztai and Seralini spoke about the Mon 863 study at a June 22 press conference in Berlin organized by Greenpeace. Both scientists are uniquely qualified to evaluate the study. Seralini studies endocrine disruptors and the impact of pesticides on health. He was one of four experts appointed to respond to the WTO challenge filed by the US against the European Union?s policy on GM food and crops. He has read all of the industry?s GM-food submissions to Europe as well as all the commentaries on the submissions. Pusztai is the leading authority in his field of protein science (lectins) and had been commissioned by the UK government in the 1990s to develop the ideal testing protocol for all GM foods. Although his protocol was supposed to be adopted by the UK government and eventually in Europe, Pusztai?s controversial finding that GM potatoes damaged the health of rats ultimately stopped the work. Pusztai has also been commissioned to evaluate all published studies on GM foods, and has analyzed most of the confidential submissions made by industry.

Both scientists have expressed alarm about the unsupported arguments that Monsanto and some European regulators use to force product approvals. Now that the Mon 863 study is available, other scientists and the public can evaluate the industry?s defense, which Pusztai and Seralini say contradict well established scientific principles. Chief among their concerns are the ways Monsanto explains away statistically significant effects.

Faulty Comparisons Hide Problems

In animal feeding studies, researchers attempt to minimize differences between the test animals and the control groups, so that only the impact of the item being analyzed will stand out. In this study therefore, the test rats ate Mon 863 and the control group ate non-GM corn from the same parent line, i.e., corn whose genetics are the same except for the insertion of the genetic material and its impact. When comparing the results of these two appropriate groups, the health impacts were unambiguous and occurred at a rate that the scientific community accepts as not due to chance. But Monsanto and their supporters in the European Food Safety Authority (EFSA) appear to throw away the accepted methods of science that have been used for decades in order to rationalize the findings.

1. Researchers used six additional control groups, which were fed commercial corn varieties with entirely different genetics. While such comparisons are appropriate for commercial studies, it is entirely inappropriate for a safety assessment, according to Pusztai. Monsanto claimed that when the changes in the test rats were compared to this much larger, irrelevant control group, many changes were no longer significant.

2. In spite of the strained logic, many results were still statistically significant when compared to these six other controls and were reported as such by the laboratory that Monsanto used to conduct the study. Monsanto therefore ignored the study?s figures and claimed that since the changes in the rats were still within a wide range of reactions that are normal for the animals, they should be considered biologically irrelevant. Using this argument, for example, they declared that a 52% decrease in reticulocytes (immature blood cells) was ?attributable to normal biological variability.? According to Pusztai, an allowance of 5% variability is the norm in food experiments. Similarly, he says that the increase in blood sugar levels by 10% ?cannot be written off as biologically insignificant, given the epidemic of diabetes.?

To put Monsanto's claims into perspective, suppose that a large number of women who were fed a carefully controlled diet had a 25% increase in breast cancer compared to matched controls on another diet. Using Monsanto's logic, the findings can be dismissed because the increase was still within the normal variability of breast cancer for the whole population.

3. In spite of the statistical slight-of-hand, several results could still not be dismissed since they were well beyond the range Monsanto had defined as normal. So the company claimed that the potentially dangerous health effects were not considered significant because the reaction among the rats was not consistent between males and females. "This is really ridiculous," says Seralini, because everyone studying cancer and endocrinology, for example, knows that there are differences between genders.

4. When even the gender defense could not be applied to a particular finding, Monsanto dismissed it since the reactions were not always dose specific. Specifically, the results observed in rats fed a diet that was 11% Mon 863 were sometimes more pronounced than results found in rats fed a 33% diet. Seralini notes that in endocrinology and toxicology research, differences are not always proportional to effects noted. A small dose of a hormone, for example, can cause a woman to ovulate, while a larger dose can make her infertile.

5. When all other excuses failed, Monsanto claimed that with such a large study, one would expect lots of results to fall in the statistically significant category purely by chance. Thus, no follow-up is required.

Seralini says, "It is dishonest not to do the tests again if you have statistical significance." Pusztai similarly asks, "What is the point of doing a study if you dismiss the results you find?" He insists that you design a study specifically so that statistical significance indicates biological significance.

In spite of the fact that Monsanto's explanations were at odds with time-honored principles of science, the European Food Standards Agency (EFSA) recommended that Mon 863 be approved. In fact, the agency's justification mimics that of Monsanto, point for point. In spite of EFSA's recommendation to approve Mon 863, the majority of the countries in the EU Council of Ministers voted not to approve the corn on July 24, 2005. But EU law requires a "qualified majority" on such a vote, and so the pro-GM European Commission is now authorized to make the decision and is expected to approve Mon 863 within a few months.

Mon 863 will not be the first approved GM food in Europe to have shown significant health effects in rats. According to Seralini, an oilseed rape (GT 73), Roundup Ready corn (NK 603), and two Bt corn varieties (Bt11 and Mon 810) all showed statistically significant problems that regulators did not pursue with follow-up research. Seralini said that the effects of the GM crops were similar to that of pesticides. Some included inflammation disorders and problems in the livers and kidneys, the two major organs involved with detoxification. Seralini is part of a research group raising money to do independent research on a GM variety he says showed more than 50 significant rat anomalies.

GM Food is Prone to Unpredicted Effects

How can a GM crop create so many significant unpredicted side effects? There are several ways. The process of gene insertion, for example, typically results in hundreds or thousands of mutations throughout the genome. Insertion also changes the amount of protein that natural genes produce (5% of the genes in one study) and can destroy natural genes altogether. The protein created by the inserted gene may also create allergies or toxins. Several studies indicate, for example, that the Bt pesticide may cause allergic or immune system effects. Furthermore, according to Monsanto's submission on Mon 863 to Australia and New Zealand, some of the foreign genetic material that was added into the corn was mutated during the insertion process. This means that the composition of the Bt protein that the corn creates is actually different than the one scientists intended.

With so many ways to create side effects, many scientists and consumer groups are demanding extensive evaluations and insist that a simple 90-day rat experiment is not competent to protect the public. In the EU, pesticide approvals require research on three types of mammals, with feeding studies ranging from 90 days to two years. Seralini points out that Bt crops create new pesticides. Mon 863, for example, is unique; it differs from the natural version of Bt pesticide in seven ways and should, according to Seralini, require at least the same level of evaluation as chemical pesticides. The same holds true for herbicide tolerant crops, which are engineered to survive large applications of weed killers such as Monsanto's Roundup. Seralini points out that these GM plants have far more herbicide residues in the edible portions and extensive toxicity tests must be performed. But the biotech industry claims that they could not afford to introduce GM crops if they had to pay for the tests normally required for pesticides in Europe. For GM crop approvals in the US, they spend even less. US authorities require only 30-day studies for the Bt plants and no safety tests whatsoever are required for herbicide tolerant varieties.

Flaws in the Mon 863 Study Should Have Caused It to be Rejected

According to Pusztai, the quality of Monsanto's study was well below that normally required for a peer reviewed publication. He says, "It is odd, therefore, that it remains the central document considered by government regulatory authorities upon which to make a decision to protect the health of European citizens."

Several features of the study appear to have been rigged to avoid finding problems. Nutritional studies, for example, typically use young, fast-growing animals, which are sensitive to toxic and nutritional effects. By using a mix of young and old animals, Monsanto's research design may have hidden serious problems. Similarly, they used rats with a huge range of starting weights. According to Pusztai, the starting weights in a rat feeding study should not vary more than 2% from the average. By contrast, the male starting weights in Monsanto's study ranged from 198.4 to 259.8 grams (or 143 to 186 grams according to the conflicting data in the study's appendix). In either case, says Pusztai, the wide range "can make it impossible to find significant differences in animal weights at the end of the experiment."

Monsanto tested the effects of two diets: in one Mon 863 constituted 33% of the rats' diet, and in the other, it was 11%. Even in the 33% group, GM corn protein comprised only about 15% of the rats' total protein. According to Pusztai, researchers should have started with the maximum amount of corn possible (while maintaining a balanced diet), and then used lower concentrations to evaluate any dose effect. (Since rats are stand-ins for humans, it is interesting to note that African aid recipients typically rely on corn for 90% of their total caloric intake.) Researchers also supplemented the corn with a commercial animal feed. Although its composition wasn't reported, it may have contained GM soy, which could have skewed the results.

The study relied on analytical methods that are half a century old and ignored powerful new methods, such as profiling techniques, DNA chips, proteomics, and others. They relied on just two observation times (week 5 and week 14), which will not give data about the intervening periods. And the short 90-day time period will miss chronic and reproductive problems, as well as problems in the next generation.

The analysis of the findings was obscured by using six irrelevant control groups fed commercial diets, as well as data from historical databases. Such comparisons are totally unacceptable in the field of nutrition. According to Pusztai, "The study should have included a control group fed the non-GM parent line, spiked with the Bt obtained from the Mon 863. If rats reacted badly to this diet, it would show that the genetic engineering process and its unpredicted side effects, and not the Bt toxin, were responsible. Pusztai says, "A second parental line spiked with a known toxin would also be useful as a positive control," to make sure the measurements are sensitive enough to detect the expected impact of the toxin. Without this, it is difficult to know if the methods were working properly.

Monsanto also defended changes in kidney weights by comparing the values with a separate study, which used different corn genetics and a different lab. According to Pusztai, this absurd inter-experimental comparison is never done and should be disregarded.

Some of the reported weight measurements were also bizarre, suggesting possible problems with animal management or faulty data. One rat dropped 53 grams in one week and gained 102 grams in the next. Some that were heaviest at the beginning of the experiment were the lightest at the end. And the rats hardly grew at all during the last four weeks.

Overall, the research paper was confusing, conflicting, and poorly reported. It failed to disclose, for example, the nutritional composition of the feed - backed up by chemical analysis - and the methods used to measure changes in the animals. Since these most basic requirements for a nutritional study were not provided, the research cannot be repeated and the results remain suspect.

Referring to the study as a whole, Pusztai says, "Nutritional scientists and leading journals would not accept these blatant inadequacies and misinterpretations."

The Politics of Science Fails to Protect the Public

When Seralini wanted to voice his concerns about the industry's safety studies, he was told by French authorities that he was legally bound to keep even his opinions confidential. A lawsuit eventually granted him the right to speak, but until June 20, 2005, biotech companies were able to keep their feeding studies hidden by claiming that they contained confidential business information. Seralini says that "No one can understand, even among EU regulators, why the composition of the blood of rats that have eaten the GM is secret." The precedent established by the German court may open the door for more biotech studies to be made public. Without disclosure, says Seralini, just a few toxicologists can make the decision without public evaluation. And too often, the decision-making body is heavily influenced by the applying company.

In his French Commission for Biomolecular Genetics (CBG), for example, the government nominates three candidates for the position of the very important "external referee." That referee studies the application and presents the relevant facts to the 18-member committee. For about ten years, the applicant companies such as Monsanto were able to choose which candidate of the three was to be the referee overseeing their products' approval process. Seralini says, "I had a big fight with the commission" over the conflict of interest. As a result, the government changed the rules, and for the Mon 863 application they allowed the president of the commission the right to choose the referee. The president, however, is a geneticist who works very closely with industry. He appointed the same person that the biotech industry had chosen in the past.

After the CBG failed to approve Monsanto's corn in 2003, the president asked for an outside scientist to re-evaluate just one of the significant differences - kidney weight. According to Seralini, the consultant ignored the blood and liver disorders entirely. And no additional research was actually conducted; the consultant simply re-examined the same data and declared the results insignificant. The commission scheduled another vote, but failed to achieve a quorum. The president ruled that a quorum would not be needed in the next meeting, and only five members showed up. The president cast the deciding vote that approved Mon 863, 3 votes to 2. The other votes in favor came from the commission's vice-president, who works at an organization that conducts agricultural research, and a scientist. According to Seralini, the scientist is a toxicologist who, oddly enough, is "always against long animal toxicity tests." In fact, he had been part of the French committee that approved Novartis (now Syngenta) E 176 corn after it had been tested for only two weeks with three cows. Actually, there were four cows at the start of the study, but one died and was removed.

The toxicologist is also on the European Food Standards Agency that endorsed Mon 863. EFSA has come under attack for including primarily pro-GM scientists. According to a November 2004 report by Friends of the Earth, "One member has direct financial links with the biotech industry and others have indirect links. . . . Two members have even appeared in promotional videos produced by the biotech industry." And several members, including the chairman, have been part of an EU-funded project with the stated goal to "facilitate market introduction of GMO's in Europe."

US Pushes its Agenda, and its Pests, on Europe

The United States government's support for biotech is no secret. In fact, it is the official policy in several US agencies to promote the industry, and some of them have attempted to push acceptance of GM crops in Europe. In the case of Mon 863, it seems that the corn is designed to solve a European problem that the US introduced. The corn is engineered with a pesticide to attack insects such as Diabrotica. According to Seralini, "Diabrotica is from a very dangerous family of insects for a wide range of crops and was absent from the European countries until the late 1990s, forbidden even in laboratories because it is very difficult to eliminate it with known chemical insecticides." He says it appears to have entered Europe from the US in large numbers during the Balkan war. Specifically, it was widespread around US military airports, whose planes were likely to have carried the pest. It has since spread primarily in Italy, France, and Germany.

According to Seralini, "Monsanto seems to have anticipated this problem." Before any infestation had been discovered, they were already field testing their corn in France in the late 1990s. Since it takes about five years of local field trials for a GM variety to be accepted in an EU nation, such early testing was necessary.

In addition to the crop pests, Europe may have also imported the US tradition of approving GM products based on faulty studies. Documents stolen from the US FDA reveal that when Monsanto's researchers intended to illustrate that their GM bovine growth hormone did not interfere with cows'; fertility, they allegedly added cows to the study that were pregnant prior to injection. An FDA whistle-blower also charged that sick cows were removed from industry studies altogether (see Seeds of Deception, http://www.newswithviews.com/HNB/Hot_New_Books.htm chapter 3).